Dr. Dale E. Bredesen, The Bredesen Protocol

Bredesen Protocol May Be Modified To Help Patients With Parkinson’s, ALS, . . . .

September 16, 2019
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In this video, Dr. Dale Bredesen, MD, suggests modifications could be made to The Bredesen Protocol for use in treating related neurodegenerative diseases such as Parkinsons, Lewy Body Dementia, Macular Degeneration, and ALS.  Dr. Bredesen addresses these questions posed by Amylee Amos, MS, RDN and founder of The Amos Institute.

  • What is the latest research by Dr. Bredesen?
  • What is Alzheimer’s Disease and how does The Bredesen Protocol treat it?
  • Why aren’t current medicines an effective treatment for Alzheimer’s Disease?
  • How do our genetics affect our health and whether we get Alzheimer’s Disease?
  • How has your wife helped you in your work?
  • How can the Amos Institute help people with The Bredesen Protocol?
  • What are the latest results of The Bredesen Protocol?
  • Does The Bredesen Protocol work for prevention of memory decline?

NOTE:  The Amos Institute works directly with your doctor to help you obtain the blood tests, genetic testing and diagnostic studies needed to obtain your ReCODE Report from Dr. Bredesen’s company. The Institute then works with you to implement your personalized program. More information may be found at https://amosinstitute.com.

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Glossary

ALZ
abbreviation for Alzheimer’s Disease
Alzheimer’s Disease
a type of dementia and a progressive disease of the brain that slowly causes impairment in memory and cognitive function. Alzheimer’s disease happens when the brain tries to protect itself from three metabolic and toxic threats:


1 - Inflammation (from infection, diet or other causes)
2 - Decline and shortage of supportive nutrients, hormones and other brain-supporting molecules
3 - Toxic substances such as metals or biotoxins (poisons produced by microbes such as molds)

The protective response causes APP (Amyloid Precursor Protein, the long molecule that protrudes from neurons) to be cut into four fragments, including amyloid-beta, that downsize the neural network and eventually destroy synapses and neurons. When the APP molecule is cut into those four pieces, it is not cut into the two pieces that nourish and maintain synapses.

Alzheimer's disease is a state of the brain in which there is an imbalance between the reorganization of synapses that have outlived their usefulness (and which the brain can stand to lose - healthy destruction) and the maintenance or creation of existing and new synapses (which the brain needs to sustain old memories and form new ones, as well as perform other cognitive functions). That imbalance  comes from too many  of the synapse- and neuron-destroying quartet  of molecules snipped from APP and too few  of the synapse- and neuron-sustaining duo  of molecules snipped from APP.

ApoE4
abbreviation for apolipoprotein E, a gene variant (allele) which is a protein that carries lipids – i.e. fats. Carrying one ApoE4 (inherited from one parent) increases your lifetime risk of Alzheimer’s to 30 percent. Carrying two copies (from both parents) increases it to 50 to 90 percent. That compares to a risk of only about 9 percent in those who carry zero copies of this allele.
APP
abbreviation for Amyloid Precursor Protein, the long molecule that protrudes from neurons

CognoscopyTM
metabolic and genetic testing that identifies cognitive decline or what may be putting you at risk for it
Dementia
umbrella term for a group of symptoms (a syndrome) without a definitive diagnosis. Dementia is a group of symptoms that affect mental cognitive tasks such as memory and reasoning. (Alzheimer’s Disease is the most common type of dementia.)
Early Alzheimer’s Disease
an early onset of Alzheimer’s Disease and uncommon form of dementia that typically affects those under the age of 65
Functional Medicine
a systems biology–based approach that focuses on identifying and addressing the root cause of disease
Heterozygous
carrying 1 copy of the ApoE4 genetic variant
Homozygous
carrying 2 copies of the ApoE4 genetic variant
Integrative Medicine
healing-oriented medicine that takes account of the whole person, including all aspects of lifestyle. It emphasizes the therapeutic relationship between practitioner and patient, is informed by evidence, and makes use of all appropriate therapies.
Ketoflex 12/3
anti-Alzheimer’s diet to include at least 12 hours of fasting, with the first 3 hours of that fast starting after dinner. It is largely plant based, but flexible as it does allow for small amounts of meat or fish.
MCI
Mild Cognitive Impairment
MCT oil
an abbreviation for medium chain triglyceride (a saturated fat) which is found in coconut oil.  Alzheimer's impairs the brain's ability to use sugar.  An MCT ketogenic diet offers an alternative energy source:  ketones.  This allows brain cells to survive better, blocking a receptor in the brain that causes memory loss.
SCI
Subjective Cognitive Impairment – a precursor to MCI (Mild Cognitive Impairment)
The Bredesen ProtocolTM
treatment program developed by Dr. Dale E. Bredesen to prevent and reverse cognitive decline (also known as The ReCODE ProtocolTM)
The ReCODE ProtocolTM
treatment program developed by Dr. Dale E. Bredesen to prevent and reverse cognitive decline (also known as The Bredesen ProtocolTM)
The ReCODE Report
computer generated report as a result of metabolic and genetic testing (CognoscopyTM)
The ReVERSE Program
customized  treatment program to reverse cognitive decline. This program starts with a ReCODE Report (a computer generated report as a result of metabolic and genetic testing called a CognoscopyTM)

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