Podcast of ALZ Survivor Julie Gregory - Success Story!
ALZ Survivor Stories, ApoE4, Diet & Lifestyle, The Bredesen Protocol

ALZ Survivor Julie G – Success Story!

December 5, 2018
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ALZ Survivor Julie G Shares Her Success Story

With research on her own, Julie discovered diet and lifestyle changes that reversed her early Alzheimer’s symptoms.  At the early age of 49, Julie was experiencing the symptoms of cognitive decline.  Genetic testing revealed she is ApoE4 homozygote.  This means Julie carries two copies of the ApoE4 gene.  As a result, her chance of developing Alzheimer’s is increased by 50-90%.

Through much research on her own, Julie began to implement dietary and lifestyle changes to improve her memory and health.   Specifically, she did this by eating only organic food, adding supplements, cutting out toxins, exercising regularly, getting plenty of sleep, and doing brain training exercises.  Subsequently, her cognition began to improve!  Important to note, this happened even before she was able to see Dr. Perlmutter and eventually Dr. Bredesen for the proper testing and treatment.

You can read more about Julie’s story and diagnosis  in Dr. Bredesen’s book, “The End of Alzheimer’s – The First Program to Prevent and Reverse Cognitive Decline.”  In detail, her daily regime is listed in the book.  With strict adherence, Julie follows her customized  program and achieves great success.   In comparison, another ALZ survivor mentioned in the book does not follow her program as closely, but is still able to maintain symptom reversal.

Important to Realize

Results can be seen even if the patient isn’t following their customized  program perfectly.  Dr. Bredesen discovered there are 36 or more causes of Alzheimer’s.  He uses the example of a roof with 36 holes.  Once enough of the holes in your roof are patched (or insults to your brain addressed), the damage (or ALZ symptoms) begin to halt and reverse.

Included in this book are many more ALZ patient stories.  Each patient utilizing The Bredesen ProtocolTM was first tested to discover what had caused their  Alzheimer’s.  Then, an individualized  program was provided for the patient to follow.  Within 4-6 months, many were seeing cognitive decline halt and symptoms reverse.

Julie’s Website and Forum – A Great Resource Providing Hope

Julie G founded the social networking website ApoE4.info. This forum includes members from all over the world who carry one or two copies of the ApoE4 gene.  This increases their lifetime risk of Alzheimer’s.  (In Julie’s case, she carries two of the genes.) Here, members can share and discuss research, and they can communicate with experts.  Many compare advice and strategies as well as contribute their own knowledge as to what has worked for them.  In his book, Dr. Bredesen states there are approximately 800 members on Julie’s site with “about 90 percent” on some variation of The Bredesen ProtocolTM. In other words, anyone can become a member. On the forum, Julie and other members receive encouragement and continue to hear repeated stories of improvement. This forum is a great resource and provides hope.

How does The Bredesen ProtocolTM work?

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Glossary


ALZ

abbreviation for Alzheimer’s Disease

Alzheimer’s Disease

a type of dementia and a progressive disease of the brain that slowly causes impairment in memory and cognitive function. Alzheimer’s disease happens when the brain tries to protect itself from three metabolic and toxic threats:

1 - Inflammation (from infection, diet or other causes)

2 - Decline and shortage of supportive nutrients, hormones and other brain-supporting molecules

3 - Toxic substances such as metals or biotoxins (poisons produced by microbes such as molds)


The protective response causes APP (Amyloid Precursor Protein, the long molecule that protrudes from neurons) to be cut into four fragments, including amyloid-beta, that downsize the neural network and eventually destroy synapses and neurons. When the APP molecule is cut into those four pieces, it is not cut into the two pieces that nourish and maintain synapses.

Alzheimer's disease is a state of the brain in which there is an imbalance between the reorganization of synapses that have outlived their usefulness (and which the brain can stand to lose - healthy destruction) and the maintenance or creation of existing and new synapses (which the brain needs to sustain old memories and form new ones, as well as perform other cognitive functions). That imbalance  comes from too many  of the synapse- and neuron-destroying quartet  of molecules snipped from APP and too few  of the synapse- and neuron-sustaining duo  of molecules snipped from APP.


ApoE4

abbreviation for apolipoprotein E, a gene variant (allele) which is a protein that carries lipids – i.e. fats. Carrying one ApoE4 (inherited from one parent) increases your lifetime risk of Alzheimer’s to 30 percent. Carrying two copies (from both parents) increases it to 50 to 90 percent. That compares to a risk of only about 9 percent in those who carry zero copies of this allele.

APP

abbreviation for Amyloid Precursor Protein, the long molecule that protrudes from neurons


Cognoscopy

metabolic and genetic testing that identifies cognitive decline or what may be putting you at risk for it

Dementia

umbrella term for a group of symptoms (a syndrome) without a definitive diagnosis. Dementia is a group of symptoms that affect mental cognitive tasks such as memory and reasoning. (Alzheimer’s Disease is the most common type of dementia.)

Early Alzheimer’s Disease

an early onset of Alzheimer’s Disease and uncommon form of dementia that typically affects those under the age of 65

Functional Medicine

a systems biology–based approach that focuses on identifying and addressing the root cause of disease

Heterozygous

carrying 1 copy of the ApoE4 genetic variant

Homozygous

carrying 2 copies of the ApoE4 genetic variant

Integrative Medicine

healing-oriented medicine that takes account of the whole person, including all aspects of lifestyle. It emphasizes the therapeutic relationship between practitioner and patient, is informed by evidence, and makes use of all appropriate therapies.

Ketoflex 12/3

anti-Alzheimer’s diet to include at least 12 hours of fasting, with the first 3 hours of that fast starting after dinner. It is largely plant based, but flexible as it does allow for small amounts of meat or fish.

MCI

Mild Cognitive Impairment

MCT oil

an abbreviation for medium chain triglyceride (a saturated fat) which is found in coconut oil.  Alzheimer's impairs the brain's ability to use sugar.  An MCT ketogenic diet offers an alternative energy source:  ketones.  This allows brain cells to survive better, blocking a receptor in the brain that causes memory loss.

SCI

Subjective Cognitive Impairment – a precursor to MCI (Mild Cognitive Impairment)

The Bredesen Protocol

treatment program developed by Dr. Dale E. Bredesen to prevent and reverse cognitive decline (also known as The ReCODE ProtocolTM)